Diabetes Mellitus Type 2 Etiology
Type 2 diabetes usually begins with insulin resistance a condition in which muscle liver and fat cells do not use insulin well.
Diabetes mellitus type 2 etiology. At first the pancreas makes more insulin to keep up with the added demand. Low in saturated fats and cholesterol. People with type 2 diabetes are said to have insulin resistance. In type 2 diabetes adult onset diabetes the pancreas makes insulin but it either doesn t produce enough or the insulin doesn t work properly.
Excess sugar can cause tingling numbness burning or pain that. Insulin is a hormone made by your pancreas that acts like a key to let blood sugar into the cells in your body for use as energy. Heart and blood vessel disease. Type 2 diabetes mellitus is the most common form of diabetes and is currently a major worldwide cause of morbidity and mortality.
Nutritionally balanced with abundant amounts of. A healthy diet for a person with diabetes is. Nine out of 10 people with diabetes have type 2. This type occurs most often in people who are over 40 years old but can occur even in childhood if there are risk factors present.
As a result your body needs more insulin to help glucose enter cells. Your pancreas makes more insulin to try to get cells to respond. Without any trans fats. This is likely to worsen given the rapidly increasing prevalence of this condition.
Reduce insulin resistance in the muscles and liver. Hyperglycemia develops as a result of pancreatic islet failure in lieu of systemic insulin resistance. Diabetes dramatically increases the risk of heart disease stroke high blood pressure and narrowing of blood. Low in total calories.
Therefore an understanding of its etiology and pathogenesis is of considerable importance. If you have type 2 diabetes cells don t respond normally to insulin. This is called insulin resistance. Description etiology diabetes mellitus type 2 dm2 formerly known as non insulin dependent diabetes and adult onset diabetes is a chronic multisystem metabolic syndrome of gradual onset characterized by an insufficient body tissue response to insulin i e insulin resistance and impaired pancreatic production of insulin.